NIH-funded research shows prenatal PFAS exposure increases maternal beta-cell dysfunction risk by 15-20% a decade postpartum, with epigenetic changes as a key mechanism. Emerging evidence connects pre...
Recent research shows prenatal PFAS exposure disrupts maternal beta cell function, increasing diabetes risk years after pregnancy, with significant public health implications. Groundbreaking study rev...
Emerging research shows prenatal PFAS exposure significantly increases risks of gestational diabetes and long-term metabolic disorders through beta cell disruption. Groundbreaking 2024 studies demonst...
Recent research shows prenatal PFAS exposure significantly increases risks of gestational diabetes and postpartum metabolic disorders, with socioeconomic disparities in exposure levels. Emerging evide...
Emerging research shows prenatal PFAS exposure significantly impacts maternal metabolic health, increasing risks of gestational diabetes and long-term beta cell dysfunction. Groundbreaking studies dem...
Recent studies confirm PFAS chemicals disrupt insulin signaling and beta-cell function, with new EPA regulations setting strict water limits while exposure routes expand through food and consumer prod...
Recent research shows prenatal PFAS exposure significantly increases risks of gestational diabetes and insulin resistance, with new data revealing widespread contamination in pregnant women. Emerging ...
New research shows prenatal PFAS exposure disrupts maternal beta-cell function for 7-9 years postpartum, with significant implications for metabolic health and policy. Groundbreaking study reveals per...
Recent research shows prenatal PFAS exposure significantly impacts maternal metabolic health, increasing diabetes risk and impairing beta-cell function decades after pregnancy. Emerging evidence demon...
Emerging research shows prenatal PFAS exposure significantly increases risks of persistent maternal metabolic disorders, including beta cell dysfunction and insulin resistance. Groundbreaking cohort s...
